参考文献/References:
[1].Lelieveld J,Evans JS,Fnais M,et al. The contribution of outdoor air pollution sources to premature mortality on a global scale[J]. Nature,2015,525(7569):367-371.
[2].Liao SH,Chiu CS,Jang LH,et al. Long-term exposures to air pollutants and risk of peripheral arterial occlusive disease:a nationwide cohort study in Taiwan[J]. Front Cardiovasc Med,2022,9:796423.
[3].江桂斌,王春霞,张爱茜. 大气细颗粒物的毒理与健康效应[M]. 北京:科学出版社,2020:3.
[4].Chen R,Jiang Y,Hu J,et al. Hourly air pollutants and acute coronary syndrome onset in 1.29 million patients[J]. Circulation,2022,145(24):1749-1760.
[5].Zhang Y,Xin L,Xiang M,et al. The molecular mechanisms of ferroptosis and its role in cardiovascular disease[J]. Biomed Pharmacother,2022,145:112423.
[6].Posadas-Sánchez R,Vargas-Alarcón G,Cardenas A,et al. Long-term exposure to ozone and fine particulate matter and risk of premature coronary artery disease:results from genetics of atherosclerotic disease Mexican study[J]. Biology(Basel),2022,11(8):1122.
[7].Rajagopalan S,Landrigan PJ. Pollution and the heart[J]. N Engl J Med,2021,385(20):1881-1892.
[8].Harmon AC,No?l A,Subramanian B,et al. Inhalation of particulate matter containing free radicals leads to decreased vascular responsiveness associated with an altered pulmonary function[J]. Am J Physiol Heart Circ Physiol,2021,321(4):H667-H683.
[9].Guo Y,Xie X,Lei L,et al. Short-term associations between ambient air pollution and stroke hospitalisations:time-series study in Shenzhen,China[J]. BMJ Open,2020,10(3):e032974.
[10].Rodins V,Lucht S,Ohlwein S,et al. Long-term exposure to ambient source-specific particulate matter and its components and incidence of cardiovascular events—The Heinz Nixdorf Recall study[J]. Environ Int,2020,142:105854.
[11].Gardner B,Ling F,Hopke PK,et al. Ambient fine particulate air pollution triggers ST-elevation myocardial infarction,but not non-ST elevation myocardial infarction:a case-crossover study[J]. Part Fibre Toxicol,2014,11:1.
[12].Du X,Jiang S,Zeng X,et al. Air pollution is associated with the development of atherosclerosis via the cooperation of CD36 and NLRP3 inflammasome in ApoE-/- mice[J]. Toxicol Lett,2018,290:123-132.
[13].Liu Y,Pan J,Fan C,et al. Short-term exposure to ambient air pollution and mortality from myocardial infarction[J]. J Am Coll Cardiol,2021,77(3):271-281.
[14].Tibuakuu M,Michos ED,Navas-Acien A,et al. Air pollution and cardiovascular disease:a focus on vulnerable populations worldwide[J]. Curr Epidemiol Rep,2018,5(4):370-378.
[15].Tilstra MH,Tiwari I,Niwa L,et al. Risk and resilience:how is the health of older adults and immigrant people living in Canada impacted by climate- and air pollution-related exposures?[J]. Int J Environ Res Public Health,2021,18(20):10575.
[16].Martin PJ,Héliot A,Trémolet G,et al. Cellular response and extracellular vesicles characterization of human macrophages exposed to fine atmospheric particulate matter[J]. Environ Pollut,2019,254(Pt A):112933.
[17].Feng L,Wei J,Liang S,et al. miR-205/IRAK2 signaling pathway is associated with urban airborne PM 2.5-induced myocardial toxicity[J]. Nanotoxicology,2020,14(9):1198-1212.
[18].Zhang J,Liang S,Ning R,et al. PM2.5-induced inflammation and lipidome alteration associated with the development of atherosclerosis based on a targeted lipidomic analysis[J]. Environ Int,2020,136:105444.
[19].Luo CM,Feng J,Zhang J,et al. 1,25-Vitamin D3 protects against cooking oil fumes-derived PM2.5-induced cell damage through its anti-inflammatory effects in cardiomyocytes[J]. Ecotoxicol Environ Saf,2019,179:249-256.
[20].Jiang Q,Zhang C,Chen S,et al. Particulate matter 2.5 induced developmental cardiotoxicity in chicken embryo and hatchling[J]. Front Pharmacol,2020,11:841.
[21]. Pei YH,Chen J,Wu X,et al. LncRNA PEAMIR inhibits apoptosis and inflammatory response in PM2.5 exposure aggravated myocardial ischemia/reperfusion injury as a competing endogenous RNA of miR-29b-3p[J]. Nanotoxicology,2020,14(5):638-653.
[22].Hu T,Zhu P,Liu Y,et al. PM2.5 induces endothelial dysfunction via activating NLRP3 inflammasome[J]. Environ Toxicol,2021,36(9):1886-1893.
[23].Ren F,Ji C,Huang Y,et al. AHR-mediated ROS production contributes to the cardiac developmental toxicity of PM2.5 in zebrafish embryos[J]. Sci Total Environ,2020,719:135097.
[24].Wang Y,Tang N,Mao M,et al. Fine particulate matter (PM2.5) promotes IgE-mediated mast cell activation through ROS/Gadd45b/JNK axis[J]. J Dermatol Sci,2021,102(1):47-57.
[25].Li H,Cai J,Chen R,et al. Particulate matter exposure and stress hormone levels:a randomized,double-blind,crossover trial of air purification[J]. Circulation,2017,136(7):618-627.
[26].Wang Q,Gan X,Li F,et al. PM2.5 exposure induces more serious apoptosis of cardiomyocytes mediated by Caspase3 through JNK/P53 pathway in hyperlipidemic rats[J]. Int J Biol Sci,2019,15(1):24-33.
[27].Xu X,Qimuge A,Wang H,et al. IRE1α/XBP1s branch of UPR links HIF1α activation to mediate ANGⅡ-dependent endothelial dysfunction under particulate matter (PM)2.5 exposure[J]. Sci Rep,2017,7(1):13507.
[28].Tian J,Shi H,Wang X,et al. The cardiotoxicity of asthmatic rats after traffic-related PM 2.5 and water-soluble components exposure mediated by endoplasmic reticulum stress and autophagy[J]. Environ Sci Pollut Res Int,2022,29(33):50704-50716.
[29].Li D,Li Z,Zhang T,et al. 2-amino-3-methylimidazo[4,5-f] quinoline triggering liver damage by inhibiting autophagy and inducing endoplasmic reticulum stress in zebrafish (Danio rerio)[J]. Toxins(Basel),2021,13(11):826.
[30].Wang Y,Tang M. PM2.5 induces autophagy and apoptosis through endoplasmic reticulum stress in human endothelial cells[J]. Sci Total Environ,2020,710:136397.
[31].Magnani ND,Marchini T,Calabró V,et al. Role of mitochondria in the redox signaling network and its outcomes in high impact inflammatory syndromes[J]. Front Endocrinol(Lausanne),2020,11:568305.
[32].Miao X,Li W,Niu B,et al. Mitochon-drial dysfunction in endothelial cells induced by airborne fine particulate matter (<2.5 μm)[J]. J Appl Toxicol,2019,39(10):1424-1432.
[33].Zeng X,Liu D,Wu W,et al. PM2.5 exposure inducing ATP alteration links with NLRP3 inflammasome activation [J]. Environ Sci Pollut Res Int,2022,29(17):24445-24456.
[34].Li R,Kou X,Geng H,et al. Mitochondrial damage:an important mechanism of ambient PM2.5 exposure-induced acute heart injury in rats[J]. J Hazard Mater,2015,287:392-401.
[35].Secker GA,Harvey NL. Regulation of VEGFR signalling in lymphatic vascular development and disease:an update[J]. Int J Mol Sci,2021,22(14):7760.
[36].Wang S,Lin Y,Zhong Y,et al. The long noncoding RNA HCG18 participates in PM2.5-mediated vascular endothelial barrier dysfunction[J]. Aging(Albany NY),2020,12(23):23960-23973.
[37].Yin J,Xia W,Li Y,et al. COX-2 mediates PM2.5-induced apoptosis and inflammation in vascular endothelial cells[J]. Am J Transl Res,2017,9(9):3967-3976.
[38].Zou L,Xiong L,Wu T,et al. NADPH oxidases regulate endothelial inflammatory injury induced by PM2.5 via AKT/eNOS/NO axis[J]. J Appl Toxicol,2022,42(5):738-749.
[39].Zhang X,Qi W,Shi Y,et al. Role of miR-145-5p/CD40 in the inflammation and apoptosis of HUVECs induced by PM2.5[J]. Toxicology,2021,464:152993.
[40].Tsou MM,Lung SC,Shen YS,et al. A community-based study on associations between PM2.5 and PM1 exposure and heart rate variability using wearable low-cost sensing devices[J]. Environ Pollut,2021,277:116761.
[41].Byun HM,Colicino E,Trevisi L,et al. Effects of air pollution and blood mitochondrial DNA methylation on markers of heart rate variability[J]. J Am Heart Assoc,2016,5(4):e003218.
[42].Ma J,Tan Q,Nie X,et al. Longitudinal relationships between polycyclic aromatic hydrocarbons exposure and heart rate variability:exploring the role of transforming growth factor-β in a general Chinese population[J]. J Hazard Mater,2022,425:127770.
[43].Cai C,Huang J,Lin Y,et al. Particulate matter 2.5 induced arrhythmogenesis mediated by TRPC3 in human induced pluripotent stem cell-derived cardiomyocytes[J]. Arch Toxicol,2019,93(4):1009-1020.
[44].Tobaldini E,Bollati V,Prado M,et al. Acute particulate matter affects cardiovascular autonomic modulation and IFN-γ methylation in healthy volunteers[J]. Environ Res,2018,161:97-103.
相似文献/References:
[1]白春兰,张军.正五聚蛋白-3:新型心血管病炎性标志物[J].心血管病学进展,2016,(1):87.[doi:10.16806/j.cnki.issn.1004-3934.2016.01.023]
BAI Chunlan,ZHANG Jun.Pentraxin-3: A Novel Inflammation Biomarker for Cardiovascular Disease[J].Advances in Cardiovascular Diseases,2016,(2):87.[doi:10.16806/j.cnki.issn.1004-3934.2016.01.023]
[2]任茂佳,贺文帅,张琪,等.围绝经期对心血管疾病相关危险因素的影响[J].心血管病学进展,2019,(6):911.[doi:10.16806/j.cnki.issn.1004-3934.2019.06.018]
REN Maojia,HE Wenshuai,ZHANG Qi,et al.Effects of Perimenopause on Cardiovascular Risk Factors[J].Advances in Cardiovascular Diseases,2019,(2):911.[doi:10.16806/j.cnki.issn.1004-3934.2019.06.018]
[3]尹琳 黄从新.JP2蛋白和心血管疾病的研究进展[J].心血管病学进展,2019,(7):1004.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.010]
YIN Lin HUANG Congxin.Research Progress of JP2 Protein and Cardiovascular Disease[J].Advances in Cardiovascular Diseases,2019,(2):1004.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.010]
[4]朱峰 汪汉 蔡琳.抗体与心血管疾病[J].心血管病学进展,2019,(7):1007.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.011]
ZHU FengWANG HanCAI Lin.Antibodies and Cardiovascular Disease[J].Advances in Cardiovascular Diseases,2019,(2):1007.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.011]
[5]邱明仙 王正龙 许官学.心肌肌球蛋白结合蛋白-C磷酸化与心血管疾病关系的研究进展[J].心血管病学进展,2019,(7):1015.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.013]
QIU MingxianWANG ZhenglongXU Guanxue.Research Progress of the Relationship Between Cardiac Myosin Binding Protein-C and Cardiovascular Disease[J].Advances in Cardiovascular Diseases,2019,(2):1015.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.013]
[6]姬楠楠 杨晓静 谢勇.单核细胞/高密度脂蛋白比值与心血管疾病的研究进展[J].心血管病学进展,2019,(7):1019.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.014]
JI Nannan YANG Xiaojing XIE Yong.Monocyte/High-density Lipoprotein Ratio and Cardiovascular Disease[J].Advances in Cardiovascular Diseases,2019,(2):1019.[doi:10.16806/j.cnki.issn.1004-3934.2019.07.014]
[7]渠海贤 李涛 程流泉.人工智能在心脏磁共振成像中的应用进展[J].心血管病学进展,2019,(5):659.[doi:10.16806/j.cnki.issn.1004-3934.2019.05.001]
[8]侯冬华 郝丽荣.长正五聚蛋白3在动脉粥样硬化和心血管疾病中作用研究的新进展[J].心血管病学进展,2019,(5):805.[doi:10.16806/j.cnki.issn.1004-3934.2019.05.035]
HOU Donghua H AO Lirong.The Study of Atherosclerosis and Cardiovascular Diseases with Pentapycin 3[J].Advances in Cardiovascular Diseases,2019,(2):805.[doi:10.16806/j.cnki.issn.1004-3934.2019.05.035]
[9]张维 张恒 康品方.外泌体在心血管疾病中的研究进展[J].心血管病学进展,2019,(5):818.[doi:10.16806/j.cnki.issn.1004-3934.2019.05.038]
Zhang WeiKang Pinfang.Exosome in Cardiovascular Diseases[J].Advances in Cardiovascular Diseases,2019,(2):818.[doi:10.16806/j.cnki.issn.1004-3934.2019.05.038]
[10]韦莹 刘书旺 李蕾 崔鸣.生长分化因子-15在心房颤动中的研究进展[J].心血管病学进展,2019,(8):1073.[doi:10.16806/j.cnki.issn.1004-3934.2019.08.001]
WEI Ying,LIU Shuwang,LI Lei,et al.Growth Differentiation Factor-15 in Development of Atrial Fibrillation[J].Advances in Cardiovascular Diseases,2019,(2):1073.[doi:10.16806/j.cnki.issn.1004-3934.2019.08.001]