参考文献/References:
[1] Pohjoismaki JL,Goffart S. The role of mitochondria in cardiac development and protection[J]. Free Radic Biol Med,2017,106:345-354.
[2] 肖丹丹,常文光,法鸿鸽,等. 2019. Parkin介导的线粒体自噬:调控心血管疾病的新机制[J].心血管病学进展,2019,40(1):131-135.
[3] Andalib S,Divani AA,Michel TM,et al. Pandora’s Box: mitochondrial defects in ischaemic heart disease and stroke[J]. Expert Rev Mol Med,2017,19:e5.
[4] Ding M,Dong Q,Liu Z,et al. Inhibition of dynamin-related protein 1 protects against myocardial ischemia-reperfusion injury in diabetic mice[J]. Cardiovasc Diabetol,2017,16(1):19
[5] Murphy MP,Hartley RC. Mitochondria as a therapeutic target for common pathologies[J]. Nat Rev Drug Discov,2018,17(12):865-886.
[6] Ong SB,Kalkhoran SB,Hernandez-Resendiz S,et al. Mitochondrial-shaping proteins in cardiac health and disease-the long and the short of it![J]. Cardiovasc Drugs Ther,2017,31(1):87-107.
[7] Chandhok G,Lazarou M,Neumann B. Structure,function,and regulation of mitofusin-2 in health and disease[J]. Biol Rev,2018,93(2):933-949.
[8] Westermann B. Bioenergetic role of mitochondrial fusion and fission[J]. Biochim Biophys Acta,2012,1817(10):1833-1838
[9] Neumann B,Hilliard MA. Loss of MEC-17 leads to microtubule instability and axonal degeneration[J]. Cell Rep,2014,6(1):93-103
[10] Osellame LD,Singh AP,Stroud DA,et al. Cooperative and independent roles of the Drp1 adaptors Mff,MiD49 and MiD51 in mitochondrial fission[J]. J Cell Sci,2016,129(11):2170-2181
[11] Otera H,Wang C,Cleland MM,et al. Mff is an essential factor for mitochondrial recruitment of Drp1 during mitochondrial fission in mammalian cells[J]. J Cell Biol,2010,191(6):1141-1158.
[12] Palmer CS,Osellame LD,Laine D,et al. MiD49 and MiD51,new components of the mitochondrial fission machinery[J]. EMBO Rep,2011,12(6):565-573
[13] Loson OC,Song Z,Chen H,et al. Fis1,Mff,MiD49 and MiD51 mediate Drp1 recruitment in mitochondrial fission[J]. Mol Biol Cell,2013,24(5):659-667
[14] Jhun BS,O-Uchi J,Adaniya SM,et al. Protein kinase D activation induces mitochondrial fragmentation and dysfunction in cardiomyocytes[J]. J Physiol,2018,596(5):827-855.
[15] Qi X,Disatnik MH,Shen N,et al. Aberrant mitochondrial fission in neurons induced by protein kinase C{delta} under oxidative stress conditions in vivo[J]. Mol Biol Cell,2011,22(2):256-265
[16] Sharp WW,Fang YH,Han M,et al. Dynamin-related protein 1(Drp1)-mediated diastolic dysfunction in myocardial ischemia-reperfusion injury:therapeutic benefits of Drp1 inhibition to reduce mitochondrial fission[J]. FASEB J,2014,28(1):316-326.
[17] Ong S,Subrayan S,Lim SY,et al. Inhibiting mitochondrial fission protects the heart against ischemia/reperfusion injury[J].Circulation,2010,121(18):2012-2022
[18] Maneechote C,Palee S,Kerdphoo S,et al. Differential temporal inhibition of mitochondrial fission by Mdivi-1 exerts effective cardioprotection in cardiac ischemia/reperfusion injury[J]. Clin Sci(Lond),2018,132(15):1669-1683.
[19] Wang JX,Jiao JQ,Li Q,et al. miR-499 regulates mitochondrial dynamics by targeting calcineurin and dynamin-related protein-1[J]. Nat Med,2011,17(1):71-78.
[20] Pennanen C,Parra V,Lopez-Crisosto C,et al. Mitochondrial fission is required for cardiomyocyte hypertrophy mediated by a Ca2+-calcineurin signaling pathway[J]. J Cell Sci,2014,127(Pt 12):2659-2671.
[21] Hasan P,Saotome M,Ikoma T,et al. Mitochondrial fission protein,dynamin-related protein 1,contributes to the promotion of hypertensive cardiac hypertrophy and fibrosis in Dahl-salt sensitive rats[J]. J Mol Cell Cardiol,2018,121:103-106.
[22] Jong CJ,Yeung J,Tseung E,et al. Leptin-induced cardiomyocyte hypertrophy is associated with enhanced mitochondrial fission[J]. Mol Cell Biochem,2019,454(1-2):33-44,
[23] Chaanine AH,Gordon RE,Kohlbrenner E,et al. Potential role of BNIP3 in cardiac remodeling,myocardial stiffness,and endoplasmic reticulum:mitochondrial calcium homeostasis in diastolic and systolic heart failure[J]. Circ Heart Fail,2013,6(3):572-583.
[24] Shirakabe A,Zhai P,Ikeda Y,et al. Drp1-dependent mitochondrial autophagy plays a protective role against pressure overload-induced mitochondrial dysfunction and heart failure[J]. Circulation,2016,133(13):1249-1263.
[25] Huang C,Lai C,Kuo C,et al. Inhibition of ERK-Drp1 signaling and mitochondria fragmentation alleviates IGF-ⅡR-induced mitochondria dysfunction during heart failure[J]. J Mol Cellular Cardiol,2018,122:58-68.
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