参考文献/References:
[1]Isselbacher EM,Preventza O,Hamilton Black J 3rd,et al. 2022 ACC/AHA Guideline for the diagnosis and management of aortic disease:a report of the American Heart Association/American College of Cardiology Joint Committee on Clinical Practice Guidelines[J]. Circulation,2022,146(24):e334-e482.
[2]Clément M,Chappell J,Raffort J,et al. Vascular smooth muscle cell plasticity and autophagy in dissecting aortic aneurysms[J]. Arterioscler Thromb Vasc Biol,2019,39(6):1149-1159.
[3]Qin HL,Bao JH,Tang JJ,et al. Arterial remodeling:the role of mitochondrial metabolism in vascular smooth muscle cells[J]. Am J Physiol Cell Physiol,2023,324(1):C183-C192.
[4]You J,Ouyang S,Xie Z,et al. The suppression of hyperlipid diet-induced ferroptosis of vascular smooth muscle cells protests against atherosclerosis independent of p53/SCL7A11/GPX4 axis[J]. J Cell Physiol,2023,238(8):1891-1908.
[5]Liu S,Huang T,Liu R,et al. Spermidine suppresses development of experimental abdominal aortic aneurysms[J]. J Am Heart Assoc,2020,9(8):e014757.
[6]Kari S,Subramanian K,Altomonte IA,et al. Programmed cell death detection methods:a systematic review and a categorical comparison[J]. Apoptosis,2022,27(7-8):482-508.
[7]Gentle IE. Supramolecular complexes in cell death and inflammation and their regulation by autophagy[J]. Front Cell Dev Biol,2019,7:73.
[8]Lu W,Zhou Y,Zeng S,et al. Loss of FoxO3a prevents aortic aneurysm formation through maintenance of VSMC homeostasis[J]. Cell Death Dis,2021,12(4):378.
[9]Li R,Wei X,Jiang DS. Protein methylation functions as the posttranslational modification switch to regulate autophagy[J]. Cell Mol Life Sci,2019,76(19):3711-3722.
[10]He Y,Yi X,Zhang Z,et al. JIB-04,a histone demethylase Jumonji C domain inhibitor,regulates phenotypic switching of vascular smooth muscle cells[J]. Clin Epigenetics,2022,14(1):101.
[11]Lei L,Zhou Y,Wang T,et al. Activation of AMP-activated protein kinase ablated the formation of aortic dissection by suppressing vascular inflammation and phenotypic switching of vascular smooth muscle cells[J]. Int Immunopharmacol,2022,112:109177.
[12]Ohno-Urabe S,Aoki H,Nishihara M,et al. Role of macrophage Socs3 in the pathogenesis of aortic dissection[J]. J Am Heart Assoc,2018.7(2):e007389.
[13]Yang K,Ren J,Li X,et al. Prevention of aortic dissection and aneurysm via an ALDH2-mediated switch in vascular smooth muscle cell phenotype[J]. Eur Heart J,2020,41(26):2442-2453.
[14]Luo C,Zhou B,Cui Y,et al. ALDH2 knockout protects against aortic dissection[J]. BMC Cardiovasc Disord,2022,22(1):443.
[15]Chen TQ,Hu N,Huo B,et al. EHMT2/G9a inhibits aortic smooth muscle cell death by suppressing autophagy activation[J]. Int J Biol Sci,2020,16(7):1252-1263.
[16]Pistritto G,Trisciuoglio D,Ceci C,et al. Apoptosis as anticancer mechanism:function and dysfunction of its modulators and targeted therapeutic strategies[J]. Aging (Albany NY),2016,8(4):603-619.
[17]D’Arcy MS. Cell death:a review of the major forms of apoptosisnecrosis and autophagy[J]. Cell Biol Int,2019,43(6):582-592.
[18]Xu X,Lai Y,Hua ZC. Apoptosis and apoptotic body:disease message and therapeutic target potentials[J]. Biosci Rep,2019.39(1):BSR20180992..
[19]Chen YY,Hsieh CY,Jayakumar T,et al. Andrographolide induces vascular smooth muscle cell apoptosis through a SHP-1-PP2A-p38MAPK-p53 cascade[J]. Sci Rep,2014,4:5651.
[20]Chakraborty A,Li Y,Zhang C,et al. Programmed cell death in aortic aneurysm and dissection:a potential therapeutic target[J]. J Mol Cell Cardiol,2022,163:67-80.
[21]Jiang N,Zhang X,Gu X,et al. Progress in understanding the role of lncRNA in programmed cell death[J]. Cell Death Discov,2021,7(1):30.
[22]Zhao G,Fu Y,Cai Z,et al. Unspliced XBP1 confers VSMC homeostasis and prevents aortic aneurysm formation via FoxO4 interaction[J]. Circ Res,2017,121(12):1331-1345.
[23]Li P,Jiang M,Li K,et al. Glutathione peroxidase 4-regulated neutrophil ferroptosis induces systemic autoimmunity[J]. Nat Immunol,2021,22(9):1107-1117.
[24]Shi F,Wang Z,Wu Q,et al. Iron deficiency promotes aortic media degeneration by activating endoplasmic reticulum stress-mediated IRE1 signaling pathway[J]. Pharmacol Res,2022,183:106366.
[25]Li N,Jiang W,Wang W,et al. Ferroptosis and its emerging roles in cardiovascular diseases[J]. Pharmacol Res,2021,166:105466.
[26]Chen Y,Fang ZM,Yi X,et al. The interaction between ferroptosis and inflammatory signaling pathways[J]. Cell Death Dis,2023,14(3):205.
[27]Li N,Yi X,He Y,et al. Targeting ferroptosis as a novel approach to alleviate aortic dissection[J]. Int J Biol Sci,2022,18(10):4118-4134.
[28]Yang M,Luo H,Yi X,et al. The epigenetic regulatory mechanisms of ferroptosis and its implications for biological processes and diseases[J]. MedComm (2020),2023,4(3):e267.
[29]Yu P,Zhang X,Liu N,et al. Pyroptosis:mechanisms and diseases[J]. Signal Transduct Target Ther,2021,6(1):128.
[30]Chen Y,He Y,Wei X,et al. Targeting regulated cell death in aortic aneurysm and dissection therapy[J]. Pharmacol Res,2022,176:106048.
[31]Billingham LK,Stoolman JS,Vasan K,et al. Mitochondrial electron transport chain is necessary for NLRP3 inflammasome activation[J]. Nat Immunol,2022,23(5):692-704.
[32]Brinkschulte R,Fu?h?ller DM,Hoss F,et al. ATP-binding and hydrolysis of human NLRP3[J]. Commun Biol,2022,5(1):1176.
[33]Ma Q. Pharmacological inhibition of the NLRP3 inflammasome:structure,molecular activation,and inhibitor-NLRP3 interaction[J]. Pharmacol Rev,2023,75(3):487-520.
[34]Coll RC,Hill JR,Day CJ,et al. MCC950 directly targets the NLRP3 ATP-hydrolysis motif for inflammasome inhibition[J]. Nat Chem Biol,2019,15(6):556-559.
[35]Li Z,Ji S,Jiang ML,et al. The regulation and modification of GSDMD signaling in diseases[J]. Front Immunol,2022,13:893912.
[36]Coll RC,Schroder K,Pelegrín P. NLRP3 and pyroptosis blockers for treating inflammatory diseases[J]. Trends Pharmacol Sci,2022,43(8):653-668.
[37]Yang Z,Shi J,Chen L,et al. Role of pyroptosis and ferroptosis in the progression of atherosclerotic plaques[J]. Front Cell Dev Biol,2022,10:811196.
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