参考文献/References:
[1] Peleli M,Flacker P,Zhuge Z,et al. Renal denervation attenuates hypertension and renal dysfunction in a model of cardiovascular and renal disease,which is associated with reduced NADPH and xanthine oxidase activity[J]. Redox Biol,2017,13:522-527.
[2] Ritchie RH,Drummond GR,Sobey CG,et al. The opposing roles of NO and oxidative stress in cardiovascular disease[J]. Pharmacol Res,2017,116:57-69.
[3] 林春,李涛,吴柱国. NADPH氧化酶在心血管疾病中的作用[J]. 海南医学,2018,29(23):3376-3379.
[4] Ahmarani L,Avedanian L,Al-Khoury J,et al. Whole-cell and nuclear NADPH oxidases levels and distribution in human endocardial endothelial,vascular smooth muscle,and vascular endothelial cells[J]. Can J Physiol Pharmacol,2013,91(1):71-79.
[5] Bedard K,Krause KH. The NOX family of ROS-generating NADPH oxidases:physiology and pathophysiology[J]. Physiol Rev,2007,87(1):245-313.
[6] Montezano AC,de Lucca CL,Persson P,et al. NADPH oxidase 5 is a pro-contractile Nox isoform and a point of cross-talk for calcium and redox signaling-implications in vascular function[J]. J Am Heart Assoc,2018,7(12):e009388.
[7] Guzik TJ,Touyz RM. Oxidative stress,inflammation,and vascular aging in hypertension[J]. Hypertension,2017,70(4):660-667.
[8] Han X,Hu Z,Chen J,et al. Associations between genetic variants of NADPH oxidase-related genes and blood pressure responses to dietary sodium intervention:the GenSalt Study[J]. Am J Hypertens,2017,30(4):427-434.
[9] Manea SA,Antonescu ML,Fenyo IM,et al. Epigenetic regulation of vascular NADPH oxidase expression and reactive oxygen species production by histone deacetylase-dependent mechanisms in experimental diabetes[J]. Redox Biol,2018,16:332-343.
[10] Stanic B,Pandey D,Fulton DJ,et al. Increased epidermal growth factor-like ligands are associated with elevated vascular nicotinamide adenine dinucleotide phosphate oxidase in a primate model of atherosclerosis[J]. Arterioscler Thromb Vasc Biol,2012,32(10):2452-2460.
[11] Jha JC,Watson A,Mathew G,et al. The emerging role of NADPH oxidase NOX5 in vascular disease[J]. Clin Sci (Lond),2017,131(10):981-990.
[12] Montezano AC,Burger D,Paravicini TM,et al. Nicotinamide adenine dinucleotide phosphate reduced oxidase 5 (Nox5) regulation by angiotensinⅡand endothelin-1 is mediated via calcium/calmodulin-dependent,rac-1-independent pathways in human endothelial cells[J]. Circ Res,2010,106(8):1363-1373.
[13] Touyz RM,Anagnostopoulou A,Camargo LL,et al. Vascular Biology of Superoxide-Generating NADPH Oxidase 5-Implications in Hypertension and Cardiovascular Disease[J]. Antioxid Redox Signal,2019,30(7):1027-1040.
[14] Montezano AC,Tsiropoulou S,Dulak-Lis M,et al. Redox signaling,Nox5 and vascular remodeling in hypertension[J]. Curr Opin Nephrol Hypertens,2015,24(5):425-433.
[15] Holterman CE,Thibodeau JF,Kennedy CR. NADPH oxidase 5 and renal disease[J]. Curr Opin Nephrol Hypertens,2015,24(1):81-87.
[16] Holterman CE,Thibodeau JF,Towaij C,et al. Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression[J]. J Am Soc Nephrol,2014,25(4):784-797.
[17] Jha JC,Banal C,Okabe J,et al. NADPH Oxidase Nox5 accelerates renal injury in diabetic nephropathy[J]. Diabetes,2017,66(10):2691-2703.
[18] Touyz RM,Anagnostopoulou A,Camargo LL,et al. Vascular biology of superoxide-generating NADPH oxidase 5-implications in hypertension and cardiovascular disease[J]. Antioxid Redox Signal,2019,30(7):1027-1040.
[19] Guzik TJ,Chen W,Gongora MC,et al. Calcium-dependent NOX5 nicotinamide adenine dinucleotide phosphate oxidase contributes to vascular oxidative stress in human coronary artery disease[J]. J Am Coll Cardiol,2008,52(22):1803-1809.
[20] Griendling KK,Sorescu D,Ushio-Fukai M. NAD(P)H oxidase:role in cardiovascular biology and disease[J]. Circ Res,2000,86(5):494-501.
[21] Banfi B,Tirone F,Durussel I,et al. Mechanism of Ca2+ activation of the NADPH oxidase 5 (NOX5)[J]. J Biol Chem ,2004,279(18):18583-18591.
[22] Hahn NE,Meischl C,Kawahara T,et al. NOX5 expression is increased in intramyocardial blood vessels and cardiomyocytes after acute myocardial infarction in humans[J]. Am J Pathol,2012,180(6):2222-2229.
[23] Manea A,Manea SA,Gan AM,et al. Human monocytes and macrophages express NADPH oxidase 5; a potential source of reactive oxygen species in atherosclerosis[J]. Biochem Biophys Res Commun,2015,461(1):172-179.
[24] Gole HK,Tharp DL,Bowles DK. Upregulation of intermediate-conductance Ca2+-activated K+ channels (KCNN4) in porcine coronary smooth muscle requires NADPH oxidase 5 (NOX5)[J]. PLoS One ,2014,9(8):e105337.
[25] Zhang Q,Malik P,Pandey D,et al. Paradoxical activation of endothelial nitric oxide synthase by NADPH oxidase[J]. Arterioscler Thromb Vasc Biol,2008,28(9):1627-1633.
[26] Schulz E,Munzel T. NOX5,a new "radical" player in human atherosclerosis?[J]. J Am Coll Cardiol,2008,52(22):1810-1812.
[27] BelAiba RS,Djordjevic T,Petry A,et al. NOX5 variants are functionally active in endothelial cells[J]. Free Radic Biol Med,2007,42(4):446-459.
[28] Siu KL,Li Q,Zhang Y,et al. NOX isoforms in the development of abdominal aortic aneurysm[J]. Redox Biol,2017,11:118-125.
[29] Guzik B,Sagan A,Ludew D,et al. Mechanisms of oxidative stress in human aortic aneurysms--association with clinical risk factors for atherosclerosis and disease severity[J].?Int J Cardiol,2013,168(3):2389-2396.
[30] Zhu C,Yu ZB,Chen XH,et al. DNA hypermethylation of the NOX5 gene in fetal ventricular septal defect[J]. Exp Ther Med,2011,2(5):1011-1015.
相似文献/References:
[1]季春影 张瑞英.心力衰竭与心肌线粒体代谢[J].心血管病学进展,2020,(1):63.[doi:10.16806/j.cnki.issn.1004-3934.2020.01.017]
JI ChunyingZHANG Ruiying.Heart Failure and Myocardial Mitochondrial Metabolism[J].Advances in Cardiovascular Diseases,2020,(12):63.[doi:10.16806/j.cnki.issn.1004-3934.2020.01.017]
[2]位晨晨,钟明.糖尿病心肌病的发病机制[J].心血管病学进展,2020,(2):135.[doi:10.16806/j.cnki.issn.1004-3934.20.02.009]
WEI Chenchen,ZHONG Ming.Pathogenesis of Diabetic Cardiomyopathy[J].Advances in Cardiovascular Diseases,2020,(12):135.[doi:10.16806/j.cnki.issn.1004-3934.20.02.009]
[3]严宁,杨春霞,马娟,等.β-谷甾醇对大鼠心肌缺血再灌注损伤和ERK1/2信号通路的影响[J].心血管病学进展,2020,(3):321.[doi:10.16806/j.cnki.issn.1004-3934.2020.03.026]
YAN Ning,YANG Chunxia,MA Juan,et al.Effects of -sitosterolon Myocardial Ischemia-reperfusion Injury and ERK1/2 Signaling Pathway in Rats[J].Advances in Cardiovascular Diseases,2020,(12):321.[doi:10.16806/j.cnki.issn.1004-3934.2020.03.026]
[4]刘家汝 关秀茹.Nrf2/ARE信号通路在动脉粥样硬化中的研究新进展[J].心血管病学进展,2020,(8):859.[doi:10.16806/j.cnki.issn.1004-3934.2020.08.019]
LIU Jiaru,GUAN Xiuru.Nrf2/ARE Signaling Pathway in Atherosclerosis[J].Advances in Cardiovascular Diseases,2020,(12):859.[doi:10.16806/j.cnki.issn.1004-3934.2020.08.019]
[5]王瑞钰,彭琳茜,李灵姣,等.硫氧还蛋白系统与高血压的研究进展[J].心血管病学进展,2020,(10):1036.[doi:10.16806/j.cnki.issn.1004-3934.2020.10.009]
WANG RuiyuPENG LinqianLI LingjiaoXUE QianWANG LiangDU WeiHUANG Jing.Thioredoxin System in Hypertension[J].Advances in Cardiovascular Diseases,2020,(12):1036.[doi:10.16806/j.cnki.issn.1004-3934.2020.10.009]
[6]李海通 闫莉.甲状腺疾病相关肺动脉高压发病机制研究进展[J].心血管病学进展,2021,(3):232.[doi:10.16806/j.cnki.issn.1004-3934.2021.03.010]
LI Haitong,YAN Li.Pathogenesis of Thyroid Disease-related Pulmonary Hypertension[J].Advances in Cardiovascular Diseases,2021,(12):232.[doi:10.16806/j.cnki.issn.1004-3934.2021.03.010]
[7]马淑青 唐其柱.氧化应激在脓毒症心肌病中的研究进展[J].心血管病学进展,2021,(2):118.[doi:10.16806/j.cnki.issn.1004-3934.2021.02.006]
MA Shuqing,TANG Qizhu.Role of Oxidative Stress in Septic Cardiomyopathy[J].Advances in Cardiovascular Diseases,2021,(12):118.[doi:10.16806/j.cnki.issn.1004-3934.2021.02.006]
[8]王晓琪 苏冠华.高尿酸血症和心力衰竭的病理生理机制、治疗和预后价值[J].心血管病学进展,2021,(9):780.[doi:10.16806/j.cnki.issn.1004-3934.2021.09.000]
WANG Xiaoqi,SU Guanhua.Pathophysiological Mechanism, Treatment and Prognostic Value of Hyperuricemia and Heart Failure[J].Advances in Cardiovascular Diseases,2021,(12):780.[doi:10.16806/j.cnki.issn.1004-3934.2021.09.000]
[9]宋雨 李耘 马丽娜.老年人衰弱和射血分数保留性心力衰竭病理生理学机制的研究进展[J].心血管病学进展,2022,(1):38.[doi:10.16806/j.cnki.issn.1004-3934.2022.01.010]
SONG Yu,LI Yun,MA Lina.Pathophysiological Mechanisms of Frailty and Heart Failure with Preserved Ejection Fraction in the Elderly[J].Advances in Cardiovascular Diseases,2022,(12):38.[doi:10.16806/j.cnki.issn.1004-3934.2022.01.010]
[10]周慧鑫 谌虎 刘志豪 周雨扬 李泽衍 许骁 陈华强 刘承哲 刘旨浩 王宇虹 王悦怡 赖燕秋 余锂镭 江洪.二甲双胍对心肌梗死后心脏功能的影响及其机制研究[J].心血管病学进展,2022,(3):265.[doi:10.16806/j.cnki.issn.1004-3934.2022.03.000]