参考文献/References:
[1] 国家心血管病中心. 中国心血管健康与疾病报告2021[M]. 北京:科学出版社,2022.
[2] Hori S,Nomura T,Sakaguchi S. Control of regulatory T cell development by the transcription factor Foxp3[J]. Science,2003,299(5609):1057-1061.
[3] Tang TT,Yuan J,Zhu ZF,et al. Regulatory T cells ameliorate cardiac remodeling after myocardial infarction[J]. Basic Res Cardiol,2012,107(1):232.
[4] Heusch G. Myocardial ischaemia-reperfusion injury and cardioprotection in perspective[J]. Nat Rev Cardiol,2020,17(12):773-789.
[5] Yellon DM,Hausenloy DJ. Myocardial reperfusion injury[J]. N Engl J Med,2007,357(11):1121-1135.
[6] Hausenloy DJ,Yellon DM. Myocardial ischemia-reperfusion injury:a neglected therapeutic target[J]. J Clin Invest,2013,123(1):92-100.
[7] Liu J,Wang H,Li J. Inflammation and inflammatory cells in myocardial infarction and reperfusion injury:a double-edged sword[J]. Clin Med Insights Cardiol,2016,10:79-84.
[8] Gershon RK,Kondo K. Cell interactions in the induction of tolerance:the role of thymic lymphocytes[J]. Immunology,1970,18(5):723-737.
[9] Fontenot JD,Gavin MA,Rudensky AY. Foxp3 programs the development and function of CD4+CD25+ regulatory T cells[J]. Nat Immunol,2003,4(4):330-336.
[10] Sharabi A,Tsokos MG,Ding Y,et al. Regulatory T cells in the treatment of disease[J]. Nat Rev Drug Discov,2018,17(11):823-844.
[11] Belkaid Y. Regulatory T cells and infection:a dangerous necessity[J]. Nat Rev Immunol,2007,7(11):875-888.
[12] Zou W. Regulatory T cells,tumour immunity and immunotherapy[J]. Nat Rev Immunol,2006,6(4):295-307.
[13] Sakaguchi S,Mikami N,Wing JB,et al. Regulatory T cells and human disease[J]. Annu Rev Immunol,2020,38:541-566.
[14] Li F,Liu D,Liu M,et al. Tregs biomimetic nanoparticle to reprogram inflammatory and redox microenvironment in infarct tissue to treat myocardial ischemia reperfusion injury in mice[J]. J Nanobiotechnology,2022,20(1):251.
[15] Saxena A,Dobaczewski M,Rai V,et al. Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function[J]. Am J Physiol Heart Circ Physiol,2014,307(8):H1233-H1242.
[16] Xia N,Jiao J,Tang TT,et al. Activated regulatory T-cells attenuate myocardial ischaemia/reperfusion injury through a CD39-dependent mechanism[J]. Clin Sci (Lond),2015,128(10):679-693.
[17] Xia N,Lu Y,Gu M,et al. A unique population of regulatory T cells in heart potentiates cardiac protection from myocardial infarction[J]. Circulation,2020,142(20):1956-1973.
[18] Dobaczewski M,Xia Y,Bujak M,et al. CCR5 signaling suppresses inflammation and reduces adverse remodeling of the infarcted heart,mediating recruitment of regulatory T cells[J]. Am J Pathol,2010,176(5):2177-2187.
[19] Lewkowicz N,Klink M,Mycko MP,et al. Neutrophil--CD4+CD25+ T regulatory cell interactions:a possible new mechanism of infectious tolerance[J]. Immunobiology,2013,218(4):455-464.
[20] Lewkowicz P,Lewkowicz N,Sasiak A,et al. Lipopolysaccharide-activated CD4+CD25+ T regulatory cells inhibit neutrophil function and promote their apoptosis and death[J]. J Immunol,2006,177(10):7155-7163.
[21] Xiao J,Yu K,Li M,et al. The IL-2/anti-IL-2 complex attenuates cardiac ischaemia-reperfusion injury through expansion of regulatory T cells[J]. Cell Physiol Biochem,2017,44(5):1810-1827.
[22] Weirather J,Hofmann UD,Beyersdorf N,et al. Foxp3+ CD4 + T cells improve healing after myocardial infarction by modulating monocyte/macrophage differentiation[J]. Circ Res,2014,115(1):55-67.
[23] Yang Z,Zingarelli B,Szabó C. Crucial role of endogenous interleukin-10 production in myocardial ischemia/reperfusion injury[J]. Circulation,2000,101(9):1019-1026.
[24] Liu YF,Chu YY,Zhang XZ,et al. TGFβ1 protects myocardium from apoptosis and oxidative damage after ischemia reperfusion[J]. Eur Rev Med Pharmacol Sci,2017,21(7):1551-1558.
[25] Ralainirina N,Poli A,Michel T,et al. Control of NK cell functions by CD4+CD25+ regulatory T cells[J]. J Leukoc Biol,2007,81(1):144-153.
[26] Cao X,Cai SF,Fehniger TA,et al. Granzyme B and perforin are important for regulatory T cell-mediated suppression of tumor clearance[J]. Immunity,2007,27(4):635-646.
[27] Borsellino G,Kleinewietfeld M,Di Mitri D,et al. Expression of ectonucleotidase CD39 by Foxp3+ Treg cells:hydrolysis of extracellular ATP and immune suppression[J]. Blood,2007,110(4):1225-1232.
[28] Gondek DC,Devries V,Nowak EC,et al. Transplantation survival is maintained by granzyme B+ regulatory cells and adaptive regulatory T cells[J]. J Immunol,2008,181(7):4752-4760.
[29] Pandiyan P,Zheng L,Ishihara S,et al. CD4+CD25+Foxp3+ regulatory T cells induce cytokine deprivation-mediated apoptosis of effector CD4 + T cells[J]. Nat Immunol,2007,8(12):1353-1362.
[30] Iikuni N,Louren?o EV,Hahn BH,et al. Cutting edge:Regulatory T cells directly suppress B cells in systemic lupus erythematosus[J]. J Immunol,2009,183(3):1518-1522.
[31] Gotot J,Dhana E,Yagita H,et al. Antigen-specific Helios-,Neuropilin-1- Tregs induce apoptosis of autoreactive B cells via PD-L1[J]. Immunol Cell Biol,2018,96(8):852-862.
[32] Gri G,Piconese S,Frossi B,et al. CD4+CD25+ regulatory T cells suppress mast cell degranulation and allergic responses through OX40-OX40L interaction[J]. Immunity,2008,29(5):771-781.
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