参考文献/References:
[1] Ju J,Song YN,Wang K. Mechanism of ferroptosis:a potential target for cardiovascular diseases treatment[J]. Aging Dis,2021,12(1):261-276.
[2] Arosio P,Ingrassia R,Cavadini P. Ferritins:a family of molecules for iron storage,antioxidation and more[J]. Biochim Biophys Acta,2009,1790(7):589-599.
[3] Kroemer G,Mari?o G,Levine B. Autophagy and the integrated stress response[J]. Mol Cell,2010,40(2):280-293.
[4] Mancias JD,Wang X,Gygi SP,et al. Quantitative proteomics identifies NCOA4 as the cargo receptor mediating ferritinophagy[J]. Nature,2014,509(7498):105-109.
[5] Kerins MJ,Ooi A. The roles of NRF2 in modulating cellular iron homeostasis[J]. Antioxid Redox Signal,2018,29(17):1756-1773.
[6] Agmon E,Solon J,Bassereau P,et al. Modeling the effects of lipid peroxidation during ferroptosis on membrane properties[J]. Sci Rep,2018,8(1):5155.
[7] Stockwell BR,Friedmann Angeli JP,Bayir H,et al. Ferroptosis:a regulated cell death nexus linking metabolism,redox biology,and disease[J]. Cell,2017,171(2):273-285.
[8] Chen X,Li X,Xu X,et al. Ferroptosis and cardiovascular disease:role of free radical-induced lipid peroxidation[J]. Free Radic Res,2021,55(4):405-415.
[9] Dixon SJ,Lemberg KM,Lamprecht MR,et al. Ferroptosis:an iron-dependent form of nonapoptotic cell death[J]. Cell,2012,149(5):1060-1072.
[10] Xie Y,Hou W,Song X,et al. Ferroptosis:process and function[J]. Cell Death Differ,2016,23(3):369-379.
[11] 周文博,孔晨飞,秦高伟,等. 铁死亡发生机制的研究进展[J]. 生物化学与生物物理进展,2018, 45(1):16-22.
[12] Sun Y,Yao X,Zhang QJ,et al. Beclin-1-dependent autophagy protects the heart during sepsis[J]. Circulation,2018,138(20):2247-2262.
[13] Mancias JD,Pontano Vaites L,Nissim S,et al. Ferritinophagy via NCOA4 is required for erythropoiesis and is regulated by iron dependent HERC2-mediated proteolysis[J]. Elife,2015,4:e10308.
[14] Gao M,Monian P,Pan Q,et al. Ferroptosis is an autophagic cell death process[J]. Cell Res,2016,26(9):1021-1032.
[15] Bellelli R,Federico G,Matte’ A,et al. NCOA4 deficiency impairs systemic iron homeostasis[J]. Cell Rep,2016,14(3):411-421.
[16] Sun X,Ou Z,Xie M,et al. HSPB1 as a novel regulator of ferroptotic cancer cell death[J]. Oncogene,2015,34(45):5617-5625.
[17] Brown CW,Amante JJ,Chhoy P,et al. Prominin2 drives ferroptosis resistance by stimulating iron export[J]. Dev Cell,2019,51(5):575-586.e4.
[18] Hou W,Xie Y,Song X,et al. Autophagy promotes ferroptosis by degradation of ferritin[J]. Autophagy,2016,12(8):1425-1428.
[19] Baba Y,Higa JK,Shimada BK,et al. Protective effects of the mechanistic target of rapamycin against excess iron and ferroptosis in cardiomyocytes[J]. Am J Physiol Heart Circ Physiol,2018,314(3):H659-H668.
[20] Sung HK,Song E,Jahng J,et al. Iron induces insulin resistance in cardiomyocytes via regulation of oxidative stress[J]. Sci Rep,2019,9(1):4668.
[21] Zhang H,Zhabyeyev P,Wang S,et al. Role of iron metabolism in heart failure:from iron deficiency to iron overload[J]. Biochim Biophys Acta Mol Basis Dis,2019,1865(7):1925-1937.
[22] Ito J,Omiya S,Rusu MC,et al. Iron derived from autophagy-mediated ferritin degradation induces cardiomyocyte death and heart failure in mice[J]. Elife,2021,10:e62174.
[23] Liu B,Zhao C,Li H,et al. Puerarin protects against heart failure induced by pressure overload through mitigation of ferroptosis[J]. Biochem Biophys Res Commun,2018,497(1):233-240.
[24] 刁长会,刘修恒. 缺血/再灌注损伤的病理生理机制[J]. 医学综述,2018,24(12):2306-2311.
[25] Bulluck H,Rosmini S,Abdel-Gadir A,et al. Residual myocardial iron following intramyocardial hemorrhage during the convalescent phase of reperfused ST-segment-elevation myocardial infarction and adverse left ventricular remodeling[J]. Circ Cardiovasc Imaging,2016,9(10):e004940.
[26] Fan Z,Cai L,Wang S,et al. Baicalin prevents myocardial ischemia/reperfusion injury through inhibiting ACSL4 mediated ferroptosis[J]. Front Pharmacol,2021,12:628988.
[27] Shan X,Lv ZY,Yin MJ,et al. The protective effect of cyanidin-3-glucoside on myocardial ischemia-reperfusion injury through ferroptosis[J]. Oxid Med Cell Longev,2021,2021:8880141.
[28] Dabkowski ER,Williamson CL,Hollander JM. Mitochondria-specific transgenic overexpression of phospholipid hydroperoxide glutathione peroxidase(GPx4) attenuates ischemia/reperfusion-associated cardiac dysfunction[J]. Free Radic Biol Med,2008,45(6):855-865.
[29] Gao M,Monian P,Quadri N,et al. Glutaminolysis and transferrin regulate ferroptosis[J]. Mol Cell,2015,59(2):298-308.
[30] Fang X,Wang H,Han D,et al. Ferroptosis as a target for protection against cardiomyopathy[J]. Proc Natl Acad Sci U S A,2019,116(7):2672-2680.
[31] Samak M,Fatullayev J,Sabashnikov A,et al. Cardiac hypertrophy:an introduction to molecular and cellular basis[J]. Med Sci Monit Basic Res,2016,22:75-79.
[32] Scimia MC,Hurtado C,Ray S,et al. APJ acts as a dual receptor in cardiac hypertrophy[J]. Nature,2012,488(7411):394-398.
[33] Tang M,Huang Z,Luo X,et al. Ferritinophagy activation and sideroflexin1-dependent mitochondria iron overload is involved in apelin-13-induced cardiomyocytes hypertrophy[J]. Free Radic Biol Med,2019,134:445-457.
[34] Zhang X,Zheng C,Gao Z,et al. SLC7A11/xCT prevents cardiac hypertrophy by inhibiting ferroptosis[J]. Cardiovasc Drugs Ther,2022,36(3):437-447.
[35] Ward-Caviness CK,Xu T,Aspelund T,et al. Improvement of myocardial infarction risk prediction via inflammation-associated metabolite biomarkers[J]. Heart,2017,103(16):1278-1285.
[36] Omiya S,Hikoso S,Imanishi Y,et al. Downregulation of ferritin heavy chain increases labile iron pool,oxidative stress and cell death in cardiomyocytes[J]. J Mol Cell Cardiol,2009,46(1):59-66.
[37] Li S,Zheng MQ,Rozanski GJ. Glutathione homeostasis in ventricular myocytes from rat hearts with chronic myocardial infarction[J]. Exp Physiol,2009,94(7):815-824.
[38] Wang Q,Wang XL,Liu HR,et al. Protective effects of cysteine analogues on acute myocardial ischemia:novel modulators of endogenous H(2)S production[J]. Antioxid Redox Signal,2010,12(10):1155-1165.
[39] Park TJ,Park JH,Lee GS,et al. Quantitative proteomic analyses reveal that GPX4 downregulation during myocardial infarction contributes to ferroptosis in cardiomyocytes[J]. Cell Death Dis,2019,10(11):835.
[40] Nishizawa H,Matsumoto M,Shindo T,et al. Ferroptosis is controlled by the coordinated transcriptional regulation of glutathione and labile iron metabolism by the transcription factor BACH1[J]. J Biol Chem,2020,295(1):69-82.
[41] Evans T. Diagnosis and management of sepsis[J]. Clin Med (Lond),2018,18(2):146-149.
[42] Arina P, Singer M. Pathophysiology of sepsis[J]. Curr Opin Anaesthesiol,2021,34(2):77-84.
[43] Li N,Wang W,Zhou H,et al. Ferritinophagy-mediated ferroptosis is involved in sepsis-induced cardiac injury[J]. Free Radic Biol Med,2020,160:303-318.
相似文献/References:
[1]袁明明 赖松青 张泽宇 吴起才.铁死亡在脓毒症心脏功能损伤中的研究进展[J].心血管病学进展,2022,(1):26.[doi:10.16806/j.cnki.issn.1004-3934.2022.01.007]
YUAN mingmingLAI SongqingZHANG ZeyuWU Qicai.Ferroptosis in Cardiac Function Impairment in Sepsis[J].Advances in Cardiovascular Diseases,2022,(9):26.[doi:10.16806/j.cnki.issn.1004-3934.2022.01.007]
[2]彭石 马茜钰 张丹 张兆元 张锦.铁死亡在心肌缺血再灌注损伤中的作用及靶向治疗研究进展[J].心血管病学进展,2022,(4):357.[doi:10.16806/j.cnki.issn.1004-3934.2022.04.017]
PENG Shi,MA Qianyu,ZHANG Dan,et al.Role and Targeted Treatment of Ferroptosis?n Myocardial Ischemia Reperfusion Injury[J].Advances in Cardiovascular Diseases,2022,(9):357.[doi:10.16806/j.cnki.issn.1004-3934.2022.04.017]
[3]彭可玲 贾晓艳 王华 刘永铭.铁死亡与心力衰竭的研究进展[J].心血管病学进展,2022,(5):432.[doi:10.16806/j.cnki.issn.1004-3934.2022.05.012]
PENG Keling,JIA Xiaoyan,WANG Hua,et al.Ferroptosis and Heart Failure[J].Advances in Cardiovascular Diseases,2022,(9):432.[doi:10.16806/j.cnki.issn.1004-3934.2022.05.012]
[4]孙悦 付松波 李亦兰.心肌细胞铁死亡及其检测方法[J].心血管病学进展,2023,(2):167.[doi:10.16806/j.cnki.issn.1004-3934.2023.02.016]
SUN Yue,FU Songbo,LI Yilan.Methods for the Detection of Ferroptosis in Cardiomyocytes[J].Advances in Cardiovascular Diseases,2023,(9):167.[doi:10.16806/j.cnki.issn.1004-3934.2023.02.016]
[5]叶宇恒 钱玲玲 王如兴 李库林.心肌缺血再灌注损伤中铁死亡的调控机制研究进展[J].心血管病学进展,2023,(5):416.[doi:10.16806/j.cnki.issn.1004-3934.2023.05.008]
YE Yuheng,QIAN Lingling,WANG Ruxing,et al.Regulatory Mechanisms of Ferroptosis in Myocardial Ischemia Reperfusion Injury[J].Advances in Cardiovascular Diseases,2023,(9):416.[doi:10.16806/j.cnki.issn.1004-3934.2023.05.008]
[6]王文杰 杨嘉馨 丁耀东 王可馨 牛佳龙 葛海龙.铁死亡在心血管疾病中的研究进展[J].心血管病学进展,2023,(5):420.[doi:10.16806/j.cnki.issn.1004-3934.2023.05.009]
WANG Wenjie,YANG Jiaxin,DING Yaodong,et al.Ferroptosis in Cardiovascular Disease[J].Advances in Cardiovascular Diseases,2023,(9):420.[doi:10.16806/j.cnki.issn.1004-3934.2023.05.009]
[7]于永丽 李艳 高奋.铁死亡在血管紧张素Ⅱ诱导的心肌肥大中的作用研究进展[J].心血管病学进展,2023,(12):1116.[doi:10.16806/j.cnki.issn.1004-3934.2023.12.014]
YU Yongli,LI Yan,GAO Fen.Ferroptosis in Angiotensin-Induced Cardiac hypertrophy[J].Advances in Cardiovascular Diseases,2023,(9):1116.[doi:10.16806/j.cnki.issn.1004-3934.2023.12.014]
[8]李心瑶 陈俊 李灼.脓毒症心肌病的发病机制研究进展[J].心血管病学进展,2024,(1):44.[doi:10.16806/j.cnki.issn.1004-3934.2023.01.012]
LI Xinyao,CHEN Jun,LI Zhuo.Pathogenesis of Septic Cardiomyopathy[J].Advances in Cardiovascular Diseases,2024,(9):44.[doi:10.16806/j.cnki.issn.1004-3934.2023.01.012]
[9]赵 珂 陈晓姝 魏希进 张 娟 刘 杨 卞雨敬 袁 杰.铁死亡的调控机制及其在蒽环类药物心脏毒性中的研究进展[J].心血管病学进展,2024,(3):261.[doi:10.16806/j.cnki.issn.1004-3934.202.03.016]
First Clinical Medical College,Shandong University of Traditional Chinese Medicine,Jinan 0000,et al.Regulatory Mechanism of Ferroptosis and Its Progress in Anthracycline-Induced Cardiotoxicity[J].Advances in Cardiovascular Diseases,2024,(9):261.[doi:10.16806/j.cnki.issn.1004-3934.202.03.016]
[10]宋自崇 王静艺 张黎军.铁死亡在阿霉素心肌病中的作用机制[J].心血管病学进展,2024,(4):307.[doi:10.16806/j.cnki.issn.1004-3934.2024.04.005]
SONG Zichong,WANG Jingyi,ZHANG Lijun.Mechanisms of ferroptosis in doxorubicin-induced cardiomyopathy[J].Advances in Cardiovascular Diseases,2024,(9):307.[doi:10.16806/j.cnki.issn.1004-3934.2024.04.005]