参考文献/References:
[1].徐林,陶波.中性粒细胞淋巴细胞比值与急性心肌梗死的相关性研究[J].中国心血管病研究,2017,15(8):257-259.
[2].Frangogiannis NG. The inflammatory response in myocardial injury, repair, and remodelling[J].Nat Rev Cardiol,2014,11(5):255-265.
[3].Saxena A, Russo I, Frangogiannis NG. Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges[J].Transl Res,2016, 167(1):152-166.
[4].Pinto AR, Ilinykh A, Ivey MJ, et al. Revisiting cardiac cellular composition[J]. Circ Res, 2015,118(3):400-409.
[5].Chen W, Spitzl A, Mathes DS, et al. Endothelial actions of ANP enhance myocardial inflammatory infiltration in the early phase after acute infarction[J].Circ Res,2016, 119 (2):237-248.
[6].Zhu M, Goetsch S, Wang Z, et al. FoxO4 promotes early inflammatory response upon myocardial infarction via endothelial arg1[J].Circ Res,2015,117(11):967-977.
[7].Entman ML, Youker K,?Shoji T, et al. Neutrophil induced oxidative injury of cardiac myocytes:a compartmented s ystem requiring CD11b/CD18-ICAM-1 adherence[J].J Clin Invest,1992, 90(4):1335-1345.
[8].Horckmans M, Ring L, Duchene J, et al. Neutrophils orchestrate post-myocardial infarction healing by polarizing macrophages towards a reparative phenotype[J]. Eur Heart J,2 016, 38(3):187-197.
[9].Mollenhauer M, Friedrichs K, Lange M, et al. Myeloperoxidase mediates postischemic arrhythmogenic ventricular remodeling[J].Circ Res,2017,121(1):56-70.
[10].Ma Y, Yabluchanskiy A, Iyer RP, et al. Temporal neutrophil polarization following m yocardial infarction[J]. Cardiovasc Res,2016,110(1):51-61.
[11].Hulsmans M, Clauss S, Xiao L, et al. Macrophages facilitateelectrical conduction in the heart[J].Cell, 2017, 169(3):510-522.e20.
[12].Wan E, Yeap XY, Dehn S, et al. Enhanced efferocytosis of apoptoti c cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction[J]. Circ Res,2013,113(8):1004-1012.
[13].Chen W, Saxena A, Li N, et al. Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts[J].Arterioscler Thromb Vasc Biol,2012, 32(11):2598-2608.
[14].Shiraishi M, Shintani Y, Shintani Y, et al. Alternatively activated macrophages determine repair of the infarcted adult murine heart[J].J Clin Invest, 2016, 126(6):2151.
[15].Howangyin KY, Zlatanova I, Pinto C, et al. Myeloid-epithelial-reproductive receptor tyrosine kinase and milk fat globule epidermal growth factor 8 coordinately improve remodeling after myocardial infarction via local delivery of vascular endothelial growth factor[J].Circulation, 2016,133(9): 826-839.
[16].Frangogiannis NG, Mendoza LH, Lindsey ML, et al. IL-10 is induced in the reperfused myocardium and may modulate the reaction to injury[J].J Immunol,2000, 165(5):2798-2808.
[17].Hofmann U, Frantz S. Role of lymphocytes in myocardial injury, healing, and remodeling after myocardial infarction[J].Circ Res,2015,116(2):354-367.
[18].Dobaczewski M, Xia Y, Bujak M, et al. CCR5 signaling suppresses inflammation and reduces adverse remodeling of the infarcted heart, mediating recruitment of regulatory t cells[J]. Am J Pathol,2010,176(5):2177-2187.
[19].Weirather J, Hofmann UDW, Beyersdorf N, et al.Foxp3+ CD4+ T cells improve healing after myocardial infarction by modulating monocyte/macrophage differentiation[J]. Circ Res,2014,115(1):55-67.
[20].Saxena A, Dobaczewski M, Rai V, et al. Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function.[J].Am J Physiol Heart Circ Physiol, 2014, 307(8):H1233.
[21].Ramjee V, Li D, Manderfield LJ, et al. Epicardial YAP/TAZ orchestrate an immunosuppressive response following myocardial infarction.[J].J Clin Invest,2017, 127(3):899-911.
[22].Somasundaram P, Ren G, Nagar H, et al. Mast cell tryptase may modulate endothelial cell phe notype in healing myocardial infarcts.[J].J Pathol,2010,205(1): 102-111.
[23].Dewald O, Ren G, Duerr GD, et al. Of mice and dogs: species-specific differences in the inflammatory response following myocardial infarction[J].Am J Pathol, 2004, 164(2):665-677.
[24].Ngkelo A, Richart A,?Kirk JA, et al. Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction [J]. J Experimental Medicine, 2016,213(7):1353-1374.
[25].Katrien VDB, Scott CL, Nindl V, et al. Myocardial infarction primes autoreactive T cells through activation of dendritic cells[J].Cell Reports,2017,18(12): 3005-3017.
[26].Shinde AV, Humeres C, Frangogiannis NG. The role of α-smooth muscle actin in fibroblast-mediated matrix contraction and remodeling[J]. Biochim Biophys Acta, 2017,1863(1):298-309.
[27].Woodall MC, Woodall BP, Gao E,et al. Cardiac fibroblast GRK2 deletion enhances contractility and remodeling following ischemia/reperfusion injury[J].Circ Res,2016,119(10):1116-1127.
[28].Frangogiannis NG.The functional pluralism of fibroblasts in the infarcted myocardium[J]. Circ Res,2016,119(10):1049-1051.
[29].Nakaya M, Watari K, Tajima M, et al. Cardiac myofibroblast engulfment of dead cells facilitates recovery after myocardial infarction [J].J Clin Invest,2016, 127(1):383-401.
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